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Medial Shoulder Instability
By Ria Acciani, MPT & David Acciani, PT

The following is a summary of an article coauthored with Dr. Sherman Canapp, Jr., DVM, MS, Diplomate ACVS. The full article and rehabilitation protocol are available on our website www.dogpt.com and in Clean Run Magazine.

 A common forelimb condition that presents in canine athletes is medial shoulder instability (MSI). This condition is similar to a “rotator cuff injury” in people. MSI may cause subtle signs of performance-related issues such as refusing tight turns, or may be as severe as a weight-bearing lameness. The joint capsule, ligaments, and surrounding muscles and tendons all contribute to the stability of the shoulder. Insult or injury to any portion of the capsuloligamentous support structures can potentially cause shoulder joint pathology resulting in pain and dysfunction. The components that are most commonly affected by MSI include the joint capsule, medial glenohumeral ligament (MGL), subscapularis tendon, supraspinatus tendon, and less commonly, the biceps tendon. Depending on the severity and chronicity of the instability, the cartilage may also be affected.

Currently, the exact cause of MSI in dogs is unknown, although it is suspected to be related to chronic repetitive activity or overuse. Overuse of the shoulder support structures leads to degeneration of the tissues and lowering the tensile strength of the tissues, which predisposes them to fraying, disruption, and eventually complete breakdown.

Based on the results of the orthopedic examination, shoulder abduction angle tests, and arthroscopic scoring, dogs are placed into one of three categories; mild, moderate, or severe. Dogs in the mild category typically have shoulder abduction angles of 30º - 40º and arthroscopic findings consisting of mild pathology; inflammation without fraying, disruption, or laxity of the MGL, subscapularis tendon, or joint capsule.


MGL fraying

Supraspinatus bulge

Dogs in the moderate category typically have abduction angles that range from 40º - 65º and arthroscopic findings consisting of fraying, disruption, and laxity of the MGL and subscapularis tendon, as well as focal synovial proliferation associated with the subscapularis tendon, and synovial hypertrophy or hyperplasia.

Dogs in the severe category typically have abduction angles greater than 65º and arthroscopic findings consisting of complete tears of the MGL and severe disruption and the supscapularis tendon and joint capsule. For this type of injury, reconstruction of the medial compartment by direct tissue reapposition and synthetic capsulorrhaphy by a medial approach may be indicated.

Radiofrequency (RF) is the preferred method for thermal capsulorrhapy in human medicine. In dogs, RF is used for treatment of tendon and ligament laxity, the joint capsule, and ablation of pathologic intra-articular structures. Following RF treatment, inflammation, repair and remodeling occur. Inflammation is a crucial step in the healing process and therefore, treatments that decrease inflammation (NSAIDs, LASER therapy, acupuncture, etc) are not recommended during the initial postoperative period (approximately 6 weeks). Treated tissue becomes weaker than pre-treatment levels before it gets stronger and must be protected for as long as three months. Following RF treatment, post-operative care includes a custom shoulder support system / hobbles (www.Dogleggs.com), strict exercise restrictions, and rehabilitation therapy.

Rehabilitation Protocol - Post-op MSI

Acute Stage: (weeks 1-3) It is important to note that conservative management is critical during this stage of healing. Exercises are geared towards stabilization of the shoulder complex to support the healing capsule.

 
ALL exercises done in hobbles except ROM and manual therapy.
Ice therapy 2-3x’s/day for the first 3 days and then only after home and clinic
  therapy/exercise sessions.
PROM, joint compressions to toes, carpus, elbow, joint mobilizations, and
 
manual treatment.
HEP of lateral raises, torso strengthening, PROM, and cervical spine stretches
  3x’s/day.
Slow leash walks may begin.
   

Sub-Acute Stage: (weeks 4-9): Protection of the healing structures remains imperative during this stage. Manual treatment remains important to reduce compensatory effects of hobble use. Treatments focus on muscular strength, maintaining ligament or musculotendinous extensibility, beginning ROM in all directions of shoulder, increasing joint mobility and improvement of the proprioceptive system. There should be minimal pain, full weight-bearing, decreased muscle spasm/guarding of the shoulder complex, full flexion of the shoulder, and full flexibility of the biceps, triceps and teres major.

 
ALL exercises done in hobbles except ROM and manual therapy.
Ice therapy may be done after therapy/exercise sessions.
Increase HEP of stabilization exercisesand torso strengthening.
Increase leash walks.
   

End-Stage: (weeks 10-12) Full ROM of shoulder complex, including scapula, should be present, with no palpable tenderness of biceps, teres major, pecs and subscapularis. Prepare for return to normal activities and sports.

 
Hobbles utilized during normal daily activity to prevent forced abduction of the
  shoulder.
Introduction of weight-bearing exercises without hobbles. Otherwise, hobble use
is still necessary.
   

Return-to-Sports Stage: (week 15-16) It is important to note that this stage can begin if healing time for your patient was normal and was able to progress to each stage without complication. Limb circumference/muscle mass should be equal or within 1/2cm of each other. No restrictions with soft tissue structures and confirmation of full healing from surgeon. Abduction angles should be considered as well.